Don’t Get Ripped off by Carbs and Get Ripped with Saturated fat

Which food item builds more muscle? (shown below)



I am sure most of you are familiar with the common belief that saturated fatty acids are the culprit of obesity and cardiovascular ailments. I am also sure most of you are familiar with the low carbohydrate craze that flooded the media about 10 years ago due to The Atkins Diet. Dr. Atkins ideas were definitely outside of the box, and while not perfect, they were on the right track. The recommended diet for optimal health, according to the original USDA food pyramid from 1992 is a very low-fat, low protein and high carbohydrate based diet. As we know, if this diet was truly nutritionally sound then we wouldn't be facing such a high obesity epidemic at present. It is pretty clear that the nutritional food pyramid is grossly outdated and the public needs to be properly educated on proper nutrition. In actuality, high carbohydrate consumption is truly the explanation for the growing number of people diagnosed with diabetes, high triglycerides, elevated blood pressure and obesity. People need to understand that carbohydrates are not nutritionally essential for survival. Amino acids and fatty acids are mandatory for proper manufacturing of new tissue repair, immune function, hair, skin, nails, cellular integrity, brain, and heart health.


Back in the day people survived off of protein and fats


For the typical desk job citizen who performs minimal daily activity, getting rid of carbohydrates in place of red meat, eggs and cheese would be in their best interest. As for extreme athletes, like Marathon runners, Football players, Swimmers, and Cyclists involved in high levels of activity, strategic carbohydrate consumption would be extremely advantageous for supplying adequate energy reserves for optimal performance. For the weight lifters, bodybuilders and fitness enthusiast's that want muscular growth and low body fat levels, trading carbohydrates for saturated fats will be extremely conducive to the goal of attaining the ideal physique. Training with weights is not extremely energy demanding and typically only lasts around an hour. Most people train in the low to medium rep range consisting of 6-12 repetitions. This will not require extreme amounts of glucose to effectively and efficiently perform. In fact, once you metabolically shift into relying on protein and fats as your primary energy substrates, you will be able to surprise yourself in the gym and become stronger without carbohydrates. When you are consuming high protein and fats and non-starchy vegetables, you will still be able to accumulate glycogen from trace carbs from protein turnover (gluconeogenesis) and vegetables. By the end of the day, your carb count could add up to 30-70 grams depending on your total

"When you are consuming high protein and fats and non-starchy vegetables, you will still be able to accumulate glycogen from trace carbs from protein turnover (gluconeogenesis) and vegetables."


caloric intake and food choices. This is why I recommend people following a low carbohydrate diet to perform cardio in the morning to burn the most body fat, however, since glucose will always be low, you could benefit from cardio at any time of the day. I also advise people to weight train in the evening as you will have built up sufficient glycogen from incidental carbohydrates eaten throughout the day. Keep in mind that you can also derive energy from your creatine phosphate stores and ATP reserves. These powerhouse energy sources come into play on your first few reps during exercise. Once these fuel substrates are depleted, then glycogen will come into play. Creatine phosphate levels will be completely restored after 4 minutes of rest.

_ATP_P = ADP and Energy

_ADP + P = ATP

_Creatine = Supply of P


(Creatine Phosphate above)

(Fatty acid oxidation above)


The coolest attribute to this way of dieting is that you will be a fat burning machine. You will constantly be mobilizing free fatty acids from the mitochondria and from intra-muscular triglycerides. Making the core of your nutrition plan comprised of lean red meat, whole eggs, cheese, and green vegetables will give you the option of selecting two different scenarios depending on your total caloric intake and energy expenditure. The first scenario is a muscle gaining phase. You will need to be in a high caloric surplus to accrue tissue. You will most likely stay leaner while in that surplus due to your favorable hormonal environment. Your serum insulin levels will be regulated and testosterone output will be optimized. The second scenario is entering a cutting or fat-loss phase. This will require a slight to moderate caloric deficit with less cardio then you would need with higher carbohydrate consumption.

Sample muscle gaining phase caloric equation:        Sample fat loss phase caloric equation:

(Bodyweight x 20 = total calories)                                  (Bodyweight x 13 = total calories)


Given the two scenarios, it is clear to understand that you will yield the best results with a protein and saturated fat based diet. You will stay leaner when gaining size and retain more tissue when reaching very low body fat levels, since ketones have been shown to exhibit a muscle sparing effect. An interesting study investigated the ideology that "A calorie is a calorie" is not accurate and The Laws of Thermodynamics do not apply regarding high fat, low carbohydrate diets. The study involved subjects who followed a low carb, higher fat diet and subjects who followed a low fat, high carbohydrate diet. The low carb group ingested a higher caloric value than subjects on a low-fat diet and lost more weight, in addition to burning more calories throughout the day. The low-carb group ate 54 extra calories a day and lost 5.8 kg while the low-fat group lost only 1.9 kg.

Another researcher discovered that people eating 300 extra calories a day on a low-carb diet lost a similar amount of weight. Another study I will mention is one regarding a 6-week very low carbohydrate diet and its effects on total and regional body composition. Results revealed some amazing details. Fat mass was decreased (-3.4 kg) while lean body mass substantially increased (+1.1 kg) at the end of the 6 week trial. Researchers believed that the fat loss can be attributed to decreased serum insulin levels and increased fatty acid oxidation due to elevated beta-hydroxybutyrate concentrations. This proves that a calorie is not always a calorie and different macronutrients influence specific metabolic actions in the body once consumed.


1.)Kennedy ARPissios POtu HRoberson RXue BAsakura KFurukawa NMarino FELiu FFKahn BBLibermann TAMaratos-Flier E, A high-fat, ketogenic diet induces a unique metabolic state in mice.Am J Physiol Endocrinol Metab. 2007 Jun;292(6):E1724-39. Epub 2007 Feb 13.


2.)Pérez-Guisado J, Ketogenic diets and weight loss: basis and effectiveness. Arch Latinoam Nutr. 2008 Jun;58(2):126-31

3.)Yancy WS JrOlsen MKGuyton JRBakst RPWestman EC, A low-carbohydrate, ketogenic diet versus a low-fat diet to treat obesity and hyperlipidemia: a randomized, controlled trial. Ann Intern Med. 2004 May 18;140(10):769-77.

4.)Feinman RD, Fein EJ. "A calorie is a calorie" violates the second law of thermodynamicsNutr J. 2004;3:9. doi: 10.1186/1475-2891-3-9

5.)Volek JS, Sharman MJ, Cómez AL, et al. Comparison of energy-restricted very-low-carbohydrate and low-fat diets on weight loss and body composition in overweight men and women. Nutr Metab (Lond) 2004

6.)Mayo Clinic (2003, November 12). High Saturated Fat, Starch Avoidance Weight Loss Diet Offers Good Preliminary Results.
ScienceDaily (Nov. 12, 2003).

7.)Denise Gellene | Times Staff Writer, Low-fat diet not tops for weight loss. Subjects on the Atkins and Mediterranean regimens lost more in an Atkins Foundation- aided study. July 17, 2008.

8.)Essen-Gustavsson, B. & Tesch, P. A. 1990. Glycogen and triglyceride
utilization in relation to muscle metabolic characteristics in men performing heavy-resistance exercise
. European Journal of Applied Physiology, 61, 5-10.

9.)Anssi H Manninen, Very-low-carbohydrate diets and preservation of muscle mass. Nutr Metab (Lond). 2006; 3: 9. Published online 2006 January 31. doi: 10.1186/1743-7075-3-9.

10.)Vazquez JA, Adibi SA. Protein sparing during treatment of obesity: ketogenic versus non-ketogenic very low calorie diet. Metabolism. 1992;41:401–14

11.)Manninen AH.
Is a calorie really a calorie? Metabolic advantage of low-carbohydrate diets. J Int Soc
Sports Nutr. 2004;1:21–26

What is the difference between DHEA 1-DHEA 4-DHEA R-DHEA and 7-DHEA?

Main effects
+ Anti-aging (youthful energy)
+ Exercise endurance
+ Recovery
+ Fat loss
+ Muscle sparing (anti-catabolic)
+ Immune system
Primary metabolite –
5-androstenediol (5-AD)
Description –
DHEA is considered "generic" or regular DHEA. It can technically be called 5-DHEA, since the double bond is located in the 5th position. 
DHEA has been popular in the life extension crowd since the 1980’s. It's typically used for its ability to support energy and general wellbeing at a dose of 50-100mg/day. (1-3) More recently, higher doses of DHEA have been used to improve body composition due to DHEA’s mild anabolic and thermogenic effects. (1, 4) This makes DHEA an excellent choice for cutting during a calorie deficient diet, since DHEA has good muscle sparing properties. (1, 17)
DHEA converts to testosterone at a rate of about 1%, however it has high conversion to 5-androstenediol, where it gets its mild androgenic and anabolic effects. (5-8) DHEA’s thermogenic properties come from its conversion to 7-oxo-DHEA. (4)
Due to DHEA’s mild androgenic effects it rarely produces hair loss or acne. Although DHEA has moderate estrogenic effects, it rarely produces gyno or undesirable estrogenic side effects.(1-3) Some more sedentary users have reported anxiety or sleeplessness with DHEA, which is likely related to the neurosteroid activity in the brain. (3) However, this effect is also reported as “motivational energy” which is a frequently reported benefit of DHEA. 
Because of DHEA’s wide range of benefits and its balanced hormonal properties it can easily be used for cutting, or to keep gains lean during a lean mass building cycle.

Main effects –
+ Muscle mass (nitrogen retention)
+ Strength
+ Blood volume (hematopoietic)
+ Recovery
+ IGF-1 & GH
Primary metabolite –
4-androstenediol (4-AD)
Description –
4-DHEA is a naturally occurring DHEA isomer. It’s structure closely resembles regular DHEA but the double bond in the 4th position dramatically changes its effects.
4-DHEA readily converts to 4-androstenediol, rather than 5-androstenediol, boosting its anabolic potency more than 2x over regular DHEA. (6-8, 11) 4-DHEA is also expected to have a higher conversion rate to testosterone compared to regular DHEA. (6, 7) The 4-DHEA also lacks the calorie burning thermogenic properties, therefore offering superior calorie retention for a bulking effect. (4) This increased anabolic potency and reduced thermogenic action will lead to noticeable gains in strength, lean tissue growth, and weight gain. 
4-DHEA will have mild estrogen conversion that can be easily balanced with a non-aromatizing steroid like androsterone or 1-DHEA. Overall gains will be similar to the original “4-AD” banned in the 2004 Steroid Control Act.
As with the other DHEA isomers, 4-DHEA is naturally occurring and non-toxic. (9) Side-effects such as oily skin or reduced fertility are considered mild and temporary. The most notable side-effect would be suppression of natural testosterone production, which makes PCT necessary after a cycle. Overall 4-DHEA is a very safe and effective lean muscle building agent.
The only downfall to 4-DHEA is its high cost due to the high dose that is required to see significant muscle building effects.
†:The “Super” (ex., "Super-5-DHEA") signifies a fatty ester attachment to the steroid molecule to assist in bioavailability.

Main effects –
+ Muscle mass (nitrogen retention)
+ Strength
+ Hardening
+ Bloat reduction
+ Recovery
Primary metabolite –
1-androstenediol (1-AD)
Description –
1-DHEA is a naturally occurring DHEA isomer which cannot convert to testosterone or estrogen — but instead converts to the non-estrogenic 1-testosterone. (10)
The total conversion to 1-testosterone is probably less than 2%. (6,7) However, 1-DHEA gets most of its effects from conversion to 1-androstenediol, which has potent muscle building and hardening effects in and of itself. (8, 11, 12) 1-androstenediol was sold as “1-AD” prior to the 2004 Steroid Control Act, and was known for producing rapid gains in lean mass with zero water retention or bloat.
1-DHEA does not convert to estrogen nor does it activate the estrogen receptor like DHEA is known to do. (13) It could be referred to as “dry DHEA”. Because of this, it will stack well with other estrogenic steroids such as 4-DHEA to produce† clean gains in muscle tissue.
As with the other DHEA isomers, 1-DHEA is naturally occurring and non-toxic. (14) Side-effects such as oily skin, reduced fertility or increased hair shedding are considered mild and temporary. The most notable side-effect would be suppression of natural testosterone production, which makes PCT necessary after a cycle. This makes 1-DHEA a very safe, legal and effective lean muscle building agent. Some users have reported lethargy with 1-DHEA. This seems to be less severe when balanced with DHEA or 4-DHEA which tend to have an anti-lethargic effect.

Main effects –
+ Fat loss
+ Weight loss
+ Muscle sparing (anti-catabolic)
+ Immune supporting
Primary metabolite –
Androstenetriol (AET)
Description –
7-oxo-DHEA is a naturally occurring metabolite of DHEA. It’s well known for its weight loss effect due to its strong thermogenic action. (4, 15, 16)
The strong thermogenic effect from 7-oxo-DHEA come from its ability to increase two thermogenic enzymes, which increases the body’s ability to burn off calories as heat. (4) Although DHEA shares a similar effect, 7-oxo-DHEA is about 2.5x more active than DHEA. (4) Human studies have shown that 7-oxo-DHEA can increase weight loss, with a majority of weight loss being actual fat tissue. (15-16) Aside from the thermogenic action, 7-oxo-DHEA also appears to be an aromatase inhibitor, which can reduce estrogen levels. 
Because of the modification in the 7th position, 7-oxo-DHEA cannot convert to testosterone or estrogen, nor does it have any androgenic or anabolic effects. (15) Because of this, it does not produce side-effects such as hair loss, acne or prostate inflammation. It also blocks the catabolic effect of cortisol, which helps to retain muscle during a low calorie diet.
Side-effects of 7-oxo-DHEA may include anxiety or sleeplessness similar to regular DHEA due to the neuro-stimulating effect. However, this is generally mild, and seems to only occur in some users. Overall 7-oxo-DHEA is very tolerable and safe at the typical 100-200mg/day dose. (15, 16)
While 7-oxo-DHEA can be useful for a cutting or weight loss protocol, it would not be wise to use during a bulking or mass building cycle. Its tendency to increase the metabolism and rate of calorie burning will make it difficult to build additional muscle mass. (4)
†:The “Super” (ex., "Super-5-DHEA") signifies a fatty ester attachment to the steroid molecule to assist in bioavailability.

Main effects –
+ Strength 
+ Hardening
+ Bloat reduction
+ Recovery
+ Anti-estrogen
+ Sex drive
+ Aggression 
Primary metabolite –
Androstanediol (5-AA)
Description –
R-DHEA is known as “Reduced DHEA” because it is a 5a-reduced metabolite of DHEA. It’s more commonly referred to as androsterone.
This naturally occurring hormone cannot convert to testosterone, but instead converts to the dihydrotestosterone (DHT). (10, 18) Similar to testosterone, DHT is responsible for masculine traits such as aggression, sex drive, and physical strength. (19,20) However, because DHT cannot convert to estrogen, it also helps reduce fat storage and water retention, making it an excellent steroid for increasing muscular hardness and vascularity. 
R-DHEA will stack well with 5-DHEA or 4-DHEA as it will help reduce water retention from under the skin, thus creating a “dry” and hard appearance. R-DHEA also has moderate anabolic properties thus allowing it to help enhance lean muscle gains. (21)
Strength gains will also be noticeable with R-DHEA due to its strong androgenic effect which will activate the central nervous system and increase muscular power. (20) This will increase explosive power with minimal bodyweight increase. The increased aggression is typically a welcomed benefit, which manifests as increased confidence and an “alpha male” feeling in sexual and social activities. 
The strong androgenic action from R-DHEA will also help support libido and erection hardness. (20) This makes R-DHEA useful to help counter the sexually suppressive effects from other steroids. The powerful androgenic effect will also block estrogenic effects, and help prevent (and reverse) gyno. (22, 23)
Side-effects from R-DHEA will be limited to androgenic side-effects such as oily skin, acne, and increased hair shedding if the user is prone. These side effects are mild and temporary for most users.
†:The “Super” (ex., "Super-5-DHEA") signifies a fatty ester attachment to the steroid molecule to assist in bioavailability.
References –
1. DHEA treatment for HIV patients: Effects on mood, androgenic and anabolic parameters. 
Rabkin, J., et al. 
Psychoneuro endocrinology. R. 25, 53-68. 2000
2. Activation of immune function by dehydroepiandrosterone (DHEA) in age- advanced men. 
Khorram O, et al.
J Gerontol 1997; 52A:M1- M7.
3. Effects of replacement dose of dehydroepiandrosterone in men and women of advancing age.
Morales AJ, et al
J Clin Endocrinol Metab. 1994 Jun;78(6):1360-7. Erratum in: J Clin Endocrinol Metab 1995 Sep;80(9):2799.
4. Ergosteroids: induction of thermogenic enzymes in liver of rats treated with steroids derived from dehydroepiandrosterone.
LARDY H, et al. 
Proc Natl Acad Sci USA 92: 6617-6619, 1995.
5. Dehydroepiandrosterone: kinetics of metabolism in normal men and women.
Bird CE et al.
J Clin Endocrinol Metab. 1978 Oct;47(4):818-22.
6. In vivo conversion of dehydroisoandrosterone to plasma androstenedione and testosterone in man.
Horton R, et al.
J Clin Endocrinol Metab. 1967 Jan;27(1):79-88.
7. In vitro metabolism of androgens in whole human blood.
Blaquier et al.
Acta Endocrinol (Copenh). 1967 Aug;55(4):697-704. No abstract available.
8. Androgens and anabolic agents
Julius A. Vida
Chemistry and pharmacology (1969) 
THOMAS et al.
J Biol Chem. 1964 Mar;239:766-72
10. Seized designer supplement named “1-Androsterone” identification as 3b-hydroxy-5a-androst-1-en-17-one and its urinary elimination.
Maria K et al.,
Steroids. 2011 Feb 16.
11. Circulating bioactive androgens in midlife women.
Chen et al.
J Clin Endocrinol Metab. 2006 Nov;91(11):4387-94. Epub 2006 Aug 29.
12. Partial agonist/antagonist properties of androstenedione and 4-androsten-3beta,17beta-diol.
Chen Fet al.
J Steroid Biochem Mol Biol. 2004 Aug;91(4-5):247-57.
13. Direct agonist/antagonist functions of dehydroepiandrosterone.
Chen et al.
Endocrinology. 2005 Nov; 146(11):4568-76. Epub 2005 Jun 30
14. Testosterone metabolism revisited: discovery of new metabolites.
Pozo, et al.
Anal Bioanal Chem. 2010 Oct;398(4):1759-70. 
15. A randomized, double blind, placebo controlled study of 3 – acetyl – 7 – oxo – dehydroepiandrosterone in healthy overweight adults. 
Kalman, D., et al. 
(2000). Curr. Ther. Res. 61, 435-442.
16. The effect of 7 – keto Naturalean on weight loss: A randomized, double blind placebo controlled trial. Zenk, J., et al. 
(2002). Curr. Ther. Res. 63, 263-272.
17. Antiglucocorticoid function of androstenetriol. Psychoneuroendocrinology 
Loria RM. Et al.
1997;22 Suppl 1:S103-8.
18. Physiological Changes in Dehydroepiandrosterone Are Not Reflected by Serum Levels of Active Androgens and Estrogens But of Their Metabolites: Intracrinology
Fernand Labrie, et al.
J Clin Endocrinol Metab. 1997 Aug;82(8):2403-9
19. Evaluation of androgen antagonism of estrogen effect by dihydrotestosterone.
Hung TT, et al. 
J Steroid Biochem. 1983 Oct;19(4):1513-20.
20. The effects of transdermal dihydrotestosterone in the aging male: a prospective, randomized, double blind study.
Kunelius P, et al.
J Clin Endocrinol Metab. 2002 Apr;87(4):1467-72.
21. Comparative activities of compounds of the androsterone-testosterone series.
Deanesly R, et al.
Biochem J. 1936 Feb;30(2):291-303.
22. Treatment of persistent pubertal gynecomastia with dihydrotestosterone heptanoate.
Eberle AJ, et al
J Pediatr. 1986 Jul;109(1):144-9.
23. Successful percutaneous dihydrotestosterone treatment of gynecomastia occurring during highly active antiretroviral therapy: four cases and a review of the literature.
Benveniste O et al.
Clin Infect Dis. 2001 Sep 15;33(6):891-3. 
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